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Introduction

Huntington’s disease (HD) is a devastating neurodegenerative disorder characterized by motor dysfunction, cognitive impairment, and psychiatric symptoms. The pathology of HD involves the progressive degeneration of striatal and cortical neurons due to the expansion of a CAG repeat in the huntingtin gene. Currently, there is no cure for HD, and available treatments only provide symptomatic relief. Therefore, there is an urgent need to develop pharmacological strategies for neuroprotection in HD.

Chapter 1: Introduction
1.1 Introduction
1.2 Background of study
1.3 Problem Statement
1.4 Objective of study
1.5 Limitation of study
1.6 Scope of study
1.7 Significance of study
1.8 Structure of the Thesis
1.9 Definition of terms

Chapter 2: Literature Review
2.1 Pathophysiology of Huntington’s disease
2.2 Current treatment options for Huntington’s disease
2.3 Pharmacological strategies for neuroprotection in neurodegenerative diseases
2.4 Emerging neuroprotective agents for Huntington’s disease
2.5 Targeting mitochondrial dysfunction in Huntington’s disease
2.6 Role of inflammation in Huntington’s disease pathogenesis
2.7 Regulation of autophagy as a neuroprotective strategy
2.8 Gene therapy approaches for Huntington’s disease
2.9 Animal models of Huntington’s disease
2.10 Clinical trials of neuroprotective agents in Huntington’s disease

Chapter 3: Research Methodology
3.1 Research design
3.2 Participants
3.3 Data collection methods
3.4 Data analysis techniques
3.5 Ethical considerations
3.6 Research timeline
3.7 Budget considerations
3.8 Potential challenges and limitations

Chapter 4: Discussion of Findings
4.1 Efficacy of neuroprotective agents in animal models of Huntington’s disease
4.2 Mechanisms of action of neuroprotective agents
4.3 Safety profiles of neuroprotective agents
4.4 Translation of preclinical findings to clinical trials
4.5 Challenges in developing effective neuroprotective strategies
4.6 Future directions for research in Huntington’s disease neuroprotection

Chapter 5: Conclusion and Summary
In conclusion, the development of pharmacological strategies for neuroprotection in Huntington’s disease is a challenging yet crucial goal. By targeting key pathogenic mechanisms and utilizing emerging neuroprotective agents, there is hope for slowing disease progression and improving patient outcomes. This thesis aims to contribute to the growing body of knowledge on neuroprotection in HD and provide insights for future research and clinical practice.

Thesis Overview: Pharmacological strategies for neuroprotection in Huntingtonʼs disease
Huntington’s disease (HD) is a progressive neurodegenerative disorder with devastating consequences for affected individuals and their families. The current standard of care for HD focuses on managing symptoms rather than slowing or halting disease progression. This thesis explores the potential of pharmacological strategies for neuroprotection in HD, with the aim of developing novel treatment approaches to improve patient outcomes.

The introductory chapter provides a comprehensive overview of HD, including its pathophysiology, treatment options, and the need for neuroprotective strategies. The literature review section delves into current research on neuroprotection in neurodegenerative diseases, highlighting key mechanisms and potential therapeutic targets for HD. The research methodology chapter outlines the design and implementation of a study to evaluate the efficacy of neuroprotective agents in HD.

The discussion of findings section presents the results of the study, including the effects of neuroprotective agents on disease progression, mechanisms of action, and safety profiles. The conclusion and summary chapter synthesizes the key findings and their implications for future research and clinical practice in HD. Overall, this thesis aims to contribute to the growing body of knowledge on neuroprotection in HD and provide insights for the development of effective treatments for this devastating disease.

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