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Introduction

Intermittent Explosive Disorder (IED) is a psychiatric condition characterized by recurrent, impulsive, and uncontrollable episodes of aggression. Individuals with IED often experience intense anger and may engage in physical or verbal aggression that is disproportionate to the triggering event. Despite the significant impact of IED on affected individuals and those around them, the underlying neurobiological mechanisms of this disorder remain poorly understood.

This thesis aims to explore novel targets in the treatment of IED, focusing on potential neurobiological pathways that may be dysregulated in individuals with this disorder. By identifying new targets for intervention, we hope to improve the treatment outcomes for individuals with IED and ultimately enhance their quality of life.

Chapter One: Introduction
1.1 Introduction
1.2 Background of Study
1.3 Problem Statement
1.4 Objective of Study
1.5 Limitation of Study
1.6 Scope of Study
1.7 Significance of Study
1.8 Structure of the Thesis
1.9 Definition of Terms

Chapter Two: Literature Review
2.1 Overview of Intermittent Explosive Disorder
2.2 Existing Treatment Approaches for IED
2.3 Neurobiological Basis of Aggression
2.4 Genetic Factors in IED
2.5 Neuroimaging Studies in IED
2.6 Inflammatory Pathways and IED
2.7 Serotonin Dysfunction in IED
2.8 Dopaminergic System in IED
2.9 Glutamatergic System in IED
2.10 Emerging Therapeutic Targets for IED

Chapter Three: Research Methodology
3.1 Research Design
3.2 Participants
3.3 Data Collection Methods
3.4 Data Analysis Techniques
3.5 Ethical Considerations
3.6 Pilot Study
3.7 Reliability and Validity
3.8 Data Management

Chapter Four: Discussion of Findings
4.1 Overview of Study Findings
4.2 Implications for Treatment
4.3 Future Research Directions
4.4 Limitations of the Study
4.5 Strengths of the Study
4.6 Comparison with Existing Literature
4.7 Practical Applications
4.8 Recommendations for Clinical Practice

Chapter Five: Conclusion and Summary
5.1 Summary of Findings
5.2 Conclusions
5.3 Contributions to the Field
5.4 Implications for Future Research
5.5 Final Thoughts

Thesis Overview on Novel Targets in Intermittent Explosive Disorder

Intermittent Explosive Disorder (IED) is a complex psychiatric condition characterized by recurrent episodes of impulsive aggression. Despite the significant impact of IED on affected individuals and their families, current treatment approaches often fail to adequately address the underlying neurobiological mechanisms of the disorder. This thesis aims to explore novel targets for intervention in IED, with a focus on potential neurobiological pathways that may be dysregulated in individuals with this disorder.

Chapter One provides an introduction to the topic, outlining the background of study, problem statement, objectives, limitations, scope, significance, structure of the thesis, and definition of key terms. Chapter Two presents a comprehensive literature review on IED, including existing treatment approaches, neurobiological basis of aggression, genetic factors, neuroimaging studies, inflammatory pathways, serotonin dysfunction, dopaminergic system, glutamatergic system, and emerging therapeutic targets for IED.

Chapter Three details the research methodology, including research design, participants, data collection methods, data analysis techniques, ethical considerations, pilot study, reliability and validity, and data management. Chapter Four presents a discussion of findings, including implications for treatment, future research directions, limitations, strengths, comparison with existing literature, practical applications, and recommendations for clinical practice. Chapter Five offers a conclusion and summary, summarizing key findings, conclusions, contributions to the field, implications for future research, and final thoughts.

Overall, this thesis aims to advance our understanding of IED and identify novel targets for intervention that may improve treatment outcomes for individuals with this disorder. By exploring new neurobiological pathways and potential therapeutic targets, we hope to pave the way for more effective and tailored interventions for individuals with IED.

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