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Introduction

The study of autism spectrum disorders (ASD) has gained significant attention in recent years due to the increasing prevalence of this neurodevelopmental disorder. ASD is characterized by deficits in social communication and interaction, as well as restricted and repetitive behaviors. Despite extensive research efforts, the underlying neurobiological mechanisms of ASD remain poorly understood. This thesis aims to contribute to the existing body of knowledge on the neurobiology of ASD, with a focus on identifying potential biomarkers and therapeutic targets for the disorder.

Chapter 1: Introduction
1.1 Introduction
1.2 Background of study
1.3 Problem Statement
1.4 Objective of study
1.5 Limitation of study
1.6 Scope of study
1.7 Significance of study
1.8 Structure of the Thesis
1.9 Definition of Terms

Chapter 2: Literature Review
2.1 Historical perspective of autism spectrum disorders
2.2 Diagnostic criteria and prevalence of ASD
2.3 Genetic and environmental factors in ASD
2.4 Neuroimaging studies in ASD
2.5 Neurochemical abnormalities in ASD
2.6 Immune dysfunction in ASD
2.7 Gut-brain axis in ASD
2.8 Animal models of ASD
2.9 Biomarkers of ASD
2.10 Therapeutic interventions for ASD

Chapter 3: Research Methodology
3.1 Research design
3.2 Participants
3.3 Data collection methods
3.4 Data analysis
3.5 Ethical considerations
3.6 Validity and reliability
3.7 Limitations of the study
3.8 Future research directions

Chapter 4: Discussion of Findings
4.1 Neurobiological mechanisms underlying ASD
4.2 Implications for diagnosis and treatment
4.3 Potential biomarkers for ASD
4.4 Therapeutic targets for ASD
4.5 Neuroplasticity in ASD
4.6 Integrating genetic and environmental factors in ASD
4.7 Novel approaches to studying ASD
4.8 Clinical implications
4.9 Recommendations for future research

Chapter 5: Conclusion and Summary
In this final chapter, the key findings of the thesis will be summarized, and the implications for the field of neurobiology of ASD will be discussed. Recommendations for future research and potential clinical applications will also be highlighted.

Thesis Overview:

Autism spectrum disorders (ASD) are a group of complex neurodevelopmental disorders characterized by deficits in social communication and interaction, as well as restricted and repetitive behaviors. Despite significant research efforts, the underlying neurobiological mechanisms of ASD remain poorly understood. This thesis aims to contribute to the existing body of knowledge on the neurobiology of ASD by reviewing the current literature, identifying potential biomarkers and therapeutic targets, and discussing the implications for diagnosis and treatment of individuals with ASD.

Chapter 1 provides an introduction to the topic, including the background of the study, problem statement, objectives, limitations, scope, significance, and structure of the thesis. Chapter 2 presents a comprehensive literature review on the historical perspective of ASD, diagnostic criteria, prevalence, genetic and environmental factors, neuroimaging studies, neurochemical abnormalities, immune dysfunction, gut-brain axis, animal models, biomarkers, and therapeutic interventions for ASD.

Chapter 3 outlines the research methodology, including the research design, participants, data collection methods, data analysis, ethical considerations, validity and reliability, limitations, and future research directions. Chapter 4 discusses the findings of the study, including the neurobiological mechanisms underlying ASD, implications for diagnosis and treatment, potential biomarkers, therapeutic targets, neuroplasticity, genetic and environmental factors, novel approaches, clinical implications, and recommendations for future research.

Finally, Chapter 5 concludes the thesis by summarizing the key findings and discussing the implications for the field of neurobiology of ASD. Recommendations for future research and potential clinical applications are also provided. This thesis aims to advance our understanding of the neurobiology of ASD and ultimately improve the lives of individuals with this complex disorder.

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