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Introduction

Multiple sclerosis (MS) is a chronic inflammatory demyelinating disease of the central nervous system (CNS) that affects over 2.3 million people worldwide. The exact etiology of MS remains unknown, but it is widely believed to be an autoimmune disorder in which the immune system attacks the myelin sheath surrounding the nerve fibers in the CNS. While the role of immune cells in the pathogenesis of MS has been heavily studied, the contribution of glial cells, particularly microglia and astrocytes, is still not well understood. This thesis aims to investigate the role of glial cells in the pathogenesis of MS, with a particular focus on their interactions with immune cells and their impact on neuroinflammation and neurodegeneration.

Chapter 1: Introduction
1.1 Introduction
1.2 Background of study
1.3 Problem Statement
1.4 Objective of study
1.5 Limitation of study
1.6 Scope of study
1.7 Significance of study
1.8 Structure of the Thesis
1.9 Definition of terms

Chapter 2: Literature Review
2.1 Overview of Multiple Sclerosis
2.2 Glial Cells in the Central Nervous System
2.3 Immune Cells in Multiple Sclerosis
2.4 Neuroinflammation in Multiple Sclerosis
2.5 Neurodegeneration in Multiple Sclerosis
2.6 Glial-Immune Cell Interactions in Multiple Sclerosis
2.7 Current Therapies for Multiple Sclerosis
2.8 Emerging Therapeutic Targets in Multiple Sclerosis
2.9 Animal Models of Multiple Sclerosis
2.10 Gaps in Current Knowledge

Chapter 3: Research Methodology
3.1 Research Design
3.2 Study Population
3.3 Data Collection Methods
3.4 Data Analysis Procedures
3.5 Ethical Considerations
3.6 Inclusion and Exclusion Criteria
3.7 Sample Size Calculation
3.8 Statistical Analysis

Chapter 4: Discussion of Findings
4.1 Role of Microglia in Multiple Sclerosis
4.2 Role of Astrocytes in Multiple Sclerosis
4.3 Glial-Immune Cell Crosstalk in Multiple Sclerosis
4.4 Impact of Glial Cells on Neuroinflammation
4.5 Impact of Glial Cells on Neurodegeneration
4.6 Therapeutic Implications
4.7 Future Directions
4.8 Limitations of the Study

Chapter 5: Conclusion and Summary
5.1 Summary of Findings
5.2 Implications of the Study
5.3 Recommendations for Future Research
5.4 Conclusion

Thesis Overview

Multiple sclerosis (MS) is a chronic inflammatory demyelinating disease of the central nervous system (CNS) that affects millions of people worldwide. While the role of immune cells in the pathogenesis of MS has been extensively studied, the contribution of glial cells, particularly microglia and astrocytes, remains unclear. This thesis aims to investigate the role of glial cells in the pathogenesis of MS, with a focus on their interactions with immune cells, impact on neuroinflammation, and role in neurodegeneration.

Chapter 1 provides an introduction to the topic, including the background of the study, problem statement, objectives, limitations, scope, significance, and structure of the thesis. Chapter 2 presents a comprehensive literature review on MS, glial cells, immune cells, neuroinflammation, neurodegeneration, glial-immune cell interactions, current therapies, emerging targets, animal models, and gaps in knowledge.

Chapter 3 outlines the research methodology, including the design, study population, data collection methods, analysis procedures, ethical considerations, criteria for inclusion/exclusion, sample size calculation, and statistical analysis. Chapter 4 discusses the findings, focusing on the role of microglia, astrocytes, glial-immune cell crosstalk, impact on neuroinflammation and neurodegeneration, therapeutic implications, future directions, and limitations.

Finally, Chapter 5 presents the conclusion and summary, summarizing the findings, discussing the implications of the study, providing recommendations for future research, and concluding the thesis. This research aims to contribute to the understanding of the pathogenesis of MS and potentially identify new therapeutic targets for the treatment of this debilitating disease.

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