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Introduction

Psoriasis is a chronic inflammatory skin disorder characterized by scaly, red patches on the skin. It affects millions of people worldwide and significantly impacts their quality of life. While the exact cause of psoriasis is still not fully understood, it is believed to involve an abnormal immune response that leads to inflammation and the rapid turnover of skin cells.

Anti-inflammatory drugs are commonly used to manage the symptoms of psoriasis and reduce inflammation. However, the molecular mechanisms of action of these drugs in psoriasis remain unclear. Understanding how these drugs work at the molecular level is crucial for the development of more effective treatments for psoriasis.

This thesis aims to investigate the molecular mechanisms of action of anti-inflammatory drugs in psoriasis. By elucidating the pathways and molecules involved in the anti-inflammatory effects of these drugs, we hope to provide insights that can inform the development of new and improved therapies for psoriasis.

Table of Contents

Chapter 1: Introduction
1.1 Introduction
1.2 Background of study
1.3 Problem Statement
1.4 Objective of study
1.5 Limitation of study
1.6 Scope of study
1.7 Significance of study
1.8 Structure of the thesis
1.9 Definition of terms

Chapter 2: Literature Review
2.1 Overview of psoriasis
2.2 Pathogenesis of psoriasis
2.3 Current treatments for psoriasis
2.4 Mechanisms of action of anti-inflammatory drugs
2.5 Role of inflammatory mediators in psoriasis
2.6 Immune response in psoriasis
2.7 Genetic factors in psoriasis
2.8 Animal models of psoriasis
2.9 Novel therapeutic targets in psoriasis
2.10 Summary of literature review

Chapter 3: Research Methodology
3.1 Study design
3.2 Selection of anti-inflammatory drugs
3.3 Cell culture experiments
3.4 Animal models
3.5 Molecular assays
3.6 Data analysis
3.7 Ethical considerations
3.8 Potential challenges
3.9 Timeline for research
3.10 Budget allocation

Chapter 4: Discussion of Findings
4.1 Effects of anti-inflammatory drugs on inflammatory pathways
4.2 Modulation of immune response by anti-inflammatory drugs
4.3 Impact of anti-inflammatory drugs on skin cell turnover
4.4 Comparison of different anti-inflammatory drugs
4.5 Insights into potential new therapeutic targets
4.6 Limitations of the study
4.7 Future directions
4.8 Implications for clinical practice
4.9 Contribution to the field
4.10 Conclusion

Chapter 5: Conclusion and Summary
5.1 Summary of findings
5.2 Implications for future research
5.3 Conclusion

Thesis Overview

Psoriasis is a chronic inflammatory skin disorder that affects millions of people worldwide. While anti-inflammatory drugs are commonly used to manage the symptoms of psoriasis, the molecular mechanisms of their action remain poorly understood. This thesis aims to investigate the molecular pathways and molecules involved in the anti-inflammatory effects of these drugs in psoriasis.

The literature review will provide an overview of psoriasis, its pathogenesis, current treatments available, and the mechanisms of action of anti-inflammatory drugs. The research methodology will outline the study design, selection of drugs, experimental techniques, and data analysis methods employed in this study.

The discussion of findings will present the effects of anti-inflammatory drugs on inflammatory pathways, immune response, and skin cell turnover in psoriasis. It will also compare different drugs, identify potential new therapeutic targets, discuss limitations of the study, and suggest future research directions.

In conclusion, this thesis aims to shed light on the molecular mechanisms of action of anti-inflammatory drugs in psoriasis, with the ultimate goal of improving treatment strategies for this debilitating skin condition.

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