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Introduction

Non-alcoholic fatty liver disease (NAFLD) is a significant public health issue that affects a large proportion of the global population. It is characterized by the accumulation of fat in the liver, which can lead to inflammation, fibrosis, and ultimately, liver failure. Despite its prevalence, the underlying mechanisms driving the development and progression of NAFLD are not completely understood.

Metabolic reprogramming, which refers to the alterations in cellular metabolism that occur in response to environmental cues, has emerged as a key player in the pathogenesis of various diseases, including cancer and metabolic disorders. In recent years, there has been growing interest in exploring the role of metabolic reprogramming in the development and progression of NAFLD.

This thesis aims to investigate the role of metabolic reprogramming in NAFLD, with a focus on understanding how changes in cellular metabolism contribute to the pathogenesis of the disease. By unraveling the mechanisms underlying metabolic reprogramming in NAFLD, this study seeks to provide insights that could potentially lead to the development of novel therapeutic strategies for the treatment of this condition.

1.1 Introduction
1.2 Background of study
1.3 Problem Statement
1.4 Objective of study
1.5 Limitation of study
1.6 Scope of study
1.7 Significance of study
1.8 Structure of the Thesis
1.9 Definition of terms

Chapter Two: Literature Review
2.1 Overview of NAFLD
2.2 Metabolic reprogramming in disease
2.3 The role of mitochondria in NAFLD
2.4 Lipid metabolism in NAFLD
2.5 Inflammation and fibrosis in NAFLD
2.6 Genetic and environmental factors in NAFLD
2.7 Current treatment strategies for NAFLD
2.8 Emerging therapeutic targets in NAFLD
2.9 Metabolomics in the study of NAFLD
2.10 Conclusion

Chapter Three: Research Methodology
3.1 Study design
3.2 Sample selection
3.3 Data collection methods
3.4 Experimental procedures
3.5 Data analysis techniques
3.6 Ethical considerations
3.7 Statistical analysis
3.8 Validation of findings

Chapter Four: Discussion of Findings
4.1 Metabolic alterations in NAFLD
4.2 Role of mitochondrial dysfunction
4.3 Lipid metabolism reprogramming
4.4 Inflammatory responses in NAFLD
4.5 Fibrotic changes in NAFLD
4.6 Impact of genetic and environmental factors
4.7 Therapeutic implications
4.8 Future directions

Chapter Five: Conclusion and Summary
5.1 Recapitulation of key findings
5.2 Implications of the study
5.3 Limitations and future research directions
5.4 Recommendations for clinical practice
5.5 Final thoughts and conclusion

Thesis Overview:

Non-alcoholic fatty liver disease (NAFLD) is a prevalent condition characterized by the accumulation of fat in the liver, which can lead to various complications such as inflammation and fibrosis. Despite its significant health burden, the underlying mechanisms driving the development and progression of NAFLD remain incompletely understood. This thesis aims to explore the role of metabolic reprogramming in the pathogenesis of NAFLD, with a focus on understanding how changes in cellular metabolism contribute to the disease. Through a comprehensive literature review, research methodology, and discussion of findings, this study seeks to provide valuable insights into the potential therapeutic targets for the treatment of NAFLD. By elucidating the mechanistic links between metabolic reprogramming and NAFLD, this research has the potential to pave the way for the development of novel treatment strategies and improve clinical outcomes for patients with this condition.

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