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Introduction

Neurodegenerative diseases are a group of disorders characterized by the progressive degeneration of neurons in the central nervous system, resulting in cognitive decline, motor dysfunction, and ultimately, death. These debilitating conditions, which include Alzheimer’s disease, Parkinson’s disease, Huntington’s disease, and amyotrophic lateral sclerosis, are a major public health concern, affecting millions of people worldwide. Despite extensive research efforts, the underlying mechanisms driving the development and progression of neurodegenerative diseases remain poorly understood.

One emerging area of research that has gained significant attention in recent years is the role of metabolic reprogramming in the pathogenesis of neurodegenerative diseases. Metabolic reprogramming refers to the alterations in cellular metabolism that occur in response to various stressors, such as nutrient deprivation, hypoxia, or oxidative stress. These metabolic changes can profoundly impact cellular function and viability, and growing evidence suggests that dysregulation of metabolic pathways may play a critical role in the development and progression of neurodegenerative diseases.

This thesis aims to explore the role of metabolic reprogramming in the pathogenesis of neurodegenerative diseases, with a focus on identifying key metabolic pathways that are dysregulated in these conditions and understanding how these alterations contribute to neuronal dysfunction and degeneration. By elucidating the metabolic changes that occur in neurodegenerative diseases, this research seeks to provide new insights into the underlying mechanisms driving these conditions and identify potential therapeutic targets for intervention.

Table of Contents

Chapter 1: Introduction
1.1 Introduction
1.2 Background of study
1.3 Problem Statement
1.4 Objective of study
1.5 Limitation of study
1.6 Scope of study
1.7 Significance of study
1.8 Structure of the Thesis
1.9 Definition of terms

Chapter 2: Literature Review
2.1 Overview of Neurodegenerative Diseases
2.2 Metabolic Reprogramming in Neurodegenerative Diseases
2.3 Key Metabolic Pathways in Neurodegenerative Diseases
2.4 Role of Mitochondrial Dysfunction in Neurodegenerative Diseases
2.5 Oxidative Stress and Neurodegeneration
2.6 Inflammation and Neurodegeneration
2.7 Therapeutic Approaches Targeting Metabolic Reprogramming
2.8 Animal Models of Metabolic Reprogramming in Neurodegenerative Diseases
2.9 Current Challenges and Future Directions
2.10 Conclusion

Chapter 3: Research Methodology
3.1 Research Design
3.2 Study Population
3.3 Data Collection Methods
3.4 Data Analysis Techniques
3.5 Ethical Considerations
3.6 Statistical Analysis
3.7 Study Variables
3.8 Research Limitations

Chapter 4: Discussion of Findings
4.1 Overview of Study Findings
4.2 Metabolic Alterations in Neurodegenerative Diseases
4.3 Implications for Disease Pathogenesis
4.4 Therapeutic Implications
4.5 Comparison with Existing Literature
4.6 Future Research Directions

Chapter 5: Conclusion
5.1 Summary of Findings
5.2 Implications for Clinical Practice
5.3 Limitations of the Study
5.4 Recommendations for Future Research
5.5 Conclusion

Thesis Overview

Neurodegenerative diseases are a group of disorders characterized by the progressive degeneration of neurons in the central nervous system, leading to cognitive decline, motor dysfunction, and ultimately, death. Despite extensive research, the underlying mechanisms driving these conditions remain poorly understood. One emerging area of research is the role of metabolic reprogramming in neurodegenerative diseases, which refers to alterations in cellular metabolism that may contribute to disease pathogenesis.

This thesis aims to explore the role of metabolic reprogramming in neurodegenerative diseases, with a focus on identifying key metabolic pathways that are dysregulated and understanding how these alterations contribute to neuronal dysfunction and degeneration. The literature review will provide an overview of neurodegenerative diseases, metabolic reprogramming, and the implications for disease pathogenesis and potential therapeutic strategies.

The research methodology will outline the study design, population, data collection methods, analysis techniques, ethical considerations, and statistical analysis. The discussion of findings will summarize the study results, implications for disease pathogenesis, therapeutic implications, and future research directions. Lastly, the conclusion will provide a summary of findings, implications for clinical practice, limitations of the study, recommendations for future research, and a concluding remark on the role of metabolic reprogramming in neurodegenerative diseases.

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