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Introduction

Drug-induced toxic epidermal necrolysis (TEN) is a severe and potentially life-threatening skin reaction that is often caused by a reaction to medications. TEN is characterized by the detachment of the skin and mucous membranes, leading to widespread blistering and peeling of the skin. While the exact mechanisms underlying drug-induced TEN are not fully understood, recent research has identified potential biomarkers that may be associated with the development and progression of this condition.

This thesis aims to provide a comprehensive overview of the current literature on drug-induced TEN biomarkers. By examining the existing research in this field, this thesis seeks to identify key biomarkers that may be useful for early detection, monitoring, and prognosis of drug-induced TEN. Furthermore, this thesis will also explore the potential implications of these biomarkers for the development of novel therapeutic strategies for this condition.

Chapter 1
1.1 Introduction
1.2 Background of study
1.3 Problem Statement
1.4 Objective of study
1.5 Limitation of study
1.6 Scope of study
1.7 Significance of study
1.8 Structure of the Thesis
1.9 Definition of terms

Chapter 2: Literature Review
2.1 Overview of Drug-induced Toxic Epidermal Necrolysis
2.2 Pathophysiology of Drug-induced TEN
2.3 Biomarkers in Drug-induced TEN: Current Understanding
2.4 Genetic Biomarkers
2.5 Immunological Biomarkers
2.6 Inflammatory Biomarkers
2.7 Diagnostic Biomarkers
2.8 Prognostic Biomarkers
2.9 Therapeutic Implications of Biomarkers
2.10 Gaps in the Literature

Chapter 3: Research Methodology
3.1 Study Design
3.2 Participant Selection
3.3 Data Collection
3.4 Data Analysis
3.5 Ethical Considerations
3.6 Validity and Reliability
3.7 Study Limitations
3.8 Future Research Directions

Chapter 4: Discussion of Findings
4.1 Overview of Study Findings
4.2 Comparison with Existing Literature
4.3 Implications for Clinical Practice
4.4 Limitations of the Study
4.5 Recommendations for Future Research

Chapter 5: Conclusion and Summary
5.1 Summary of Key Findings
5.2 Implications for Drug-induced TEN Biomarker Research
5.3 Conclusion
5.4 Recommendations for Clinical Practice
5.5 Future Research Directions

Thesis Overview:

Drug-induced toxic epidermal necrolysis (TEN) is a severe and potentially fatal skin reaction that is often triggered by adverse reactions to medications. Despite advances in medical science, the exact mechanisms underlying drug-induced TEN remain poorly understood. This thesis aims to address this gap in knowledge by investigating the potential role of biomarkers in the early detection, monitoring, and prognosis of drug-induced TEN.

The literature review in Chapter 2 provides an overview of the current understanding of drug-induced TEN, including its pathophysiology and the role of biomarkers in this condition. By synthesizing existing research on genetic, immunological, and inflammatory biomarkers in drug-induced TEN, this chapter highlights key gaps in the literature and sets the stage for the empirical study presented in Chapter 3.

Chapter 3 outlines the research methodology employed in this thesis, including the study design, participant selection criteria, data collection methods, and data analysis techniques. By following a rigorous and systematic approach, this study aims to generate reliable and valid findings that contribute to the growing body of knowledge on drug-induced TEN biomarkers.

The discussion of findings in Chapter 4 presents a detailed analysis of the empirical results obtained from the study. By comparing and contrasting these findings with the existing literature, this chapter sheds light on the potential implications of biomarkers for clinical practice and offers recommendations for future research in this area.

In conclusion, Chapter 5 summarizes the key findings of this thesis and discusses their implications for drug-induced TEN biomarker research. By highlighting the significance of biomarkers in the diagnosis, management, and treatment of this condition, this thesis aims to advance our understanding of drug-induced TEN and pave the way for the development of novel therapeutic strategies in the future.

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