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Table of Contents:

Chapter 1: Introduction
1.1 Background of Huntingtonʼs Disease
1.2 Overview of Animal Models in Huntingtonʼs Disease Research
1.3 Purpose of the Study
1.4 Research Questions
1.5 Significance of the Study

Chapter 2: Literature Review
2.1 History and Clinical Features of Huntingtonʼs Disease
2.2 Genetics and Molecular Pathology of Huntingtonʼs Disease
2.3 Animal Models Used in Huntingtonʼs Disease Research
2.4 Neurobiological Insights from Animal Models
2.5 Current Challenges and Future Directions

Chapter 3: Research Methodology
3.1 Selection of Animal Models
3.2 Experimental Design
3.3 Data Collection and Analysis
3.4 Ethical Considerations
3.5 Limitations of the Study

Chapter 4: Discussion of Findings
4.1 Behavioral and motor deficits in animal models
4.2 Neurochemical changes in Huntingtonʼs Disease
4.3 Structural and functional alterations in the brain
4.4 Potential therapeutic interventions
4.5 Implications for future research

Chapter 5: Conclusion and Summary
5.1 Summary of Findings
5.2 Implications of the Study
5.3 Recommendations for Future Research
5.4 Conclusion

Overview of Thesis “Neurobiology of Huntingtonʼs Disease: Insights from Animal Models”:

Huntingtonʼs Disease is a devastating neurodegenerative disorder characterized by progressive motor and cognitive impairments. The genetic mutation responsible for the disease has been well characterized, but the underlying neurobiology and pathophysiology are still not fully understood. Animal models have played a crucial role in advancing our understanding of Huntingtonʼs Disease, providing valuable insights into the molecular, cellular, and behavioral changes associated with the disorder.

This thesis aims to review the current literature on the neurobiology of Huntingtonʼs Disease, with a focus on insights gained from animal models. The research methodology includes a comprehensive review of relevant studies, analysis of behavioral and neurochemical data from animal models, and discussion of potential therapeutic strategies. The limitations of the study are also acknowledged, including the challenges of translating findings from animal models to human patients.

The findings of this study will contribute to our understanding of the pathophysiology of Huntingtonʼs Disease and may inform the development of novel treatment approaches. Overall, this thesis seeks to advance our knowledge of Huntingtonʼs Disease and provide a foundation for future research in this field.

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