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Introduction:

In recent years, the field of non-coding RNA (ncRNA) research has gained significant attention due to its crucial regulatory roles in gene expression and cellular processes. Long non-coding RNAs (lncRNAs) are a subset of ncRNAs that have been found to play key roles in various biological processes, including development, differentiation, and disease. One area of growing interest is the involvement of lncRNAs in drug-induced cardiotoxicity, a serious side effect of many therapeutic drugs.

This thesis aims to explore the role of lncRNAs in the regulation of drug-induced cardiotoxicity. By investigating the interactions between lncRNAs and specific drugs, we hope to gain a better understanding of the molecular mechanisms underlying drug-induced cardiotoxicity and potentially identify new therapeutic targets for its prevention and treatment.

Chapter 1: Introduction
1.1 Introduction
1.2 Background of study
1.3 Problem Statement
1.4 Objective of study
1.5 Limitation of study
1.6 Scope of study
1.7 Significance of study
1.8 Structure of the Thesis
1.9 Definition of terms

Chapter 2: Literature Review
2.1 Overview of drug-induced cardiotoxicity
2.2 Role of lncRNAs in gene regulation
2.3 Regulatory mechanisms of lncRNAs in cardiotoxicity
2.4 Examples of lncRNAs involved in drug-induced cardiotoxicity
2.5 Current therapeutic strategies for drug-induced cardiotoxicity
2.6 Challenges in studying lncRNAs in cardiotoxicity
2.7 Emerging trends in lncRNA research
2.8 Potential of lncRNAs as therapeutic targets
2.9 Summary of current knowledge gaps
2.10 Justification for further research

Chapter 3: Research Methodology
3.1 Study design
3.2 Selection of study participants
3.3 Data collection methods
3.4 RNA isolation and lncRNA profiling
3.5 Bioinformatic analysis
3.6 Validation of results
3.7 Statistical analysis
3.8 Ethical considerations

Chapter 4: Discussion of Findings
4.1 Profile of lncRNAs in drug-induced cardiotoxicity
4.2 Molecular mechanisms underlying lncRNA-mediated cardiotoxicity
4.3 Comparison with existing literature
4.4 Implications for clinical practice
4.5 Future research directions
4.6 Strengths and limitations of the study
4.7 Conclusion

Chapter 5: Conclusion and Summary
5.1 Summary of key findings
5.2 Contributions to the field
5.3 Practical implications
5.4 Recommendations for future research
5.5 Conclusion

Thesis Overview:

Drug-induced cardiotoxicity is a serious side effect of many commonly used therapeutic drugs, leading to significant morbidity and mortality. Understanding the molecular mechanisms underlying this phenomenon is crucial for the development of effective prevention and treatment strategies. In recent years, lncRNAs have emerged as important regulators of gene expression and cellular processes, providing new insights into the pathophysiology of various diseases, including drug-induced cardiotoxicity.

This thesis aims to explore the role of lncRNAs in the regulation of drug-induced cardiotoxicity. Through a comprehensive review of the literature, analysis of lncRNA profiles in drug-exposed cardiac cells, and validation studies, we seek to identify key lncRNAs involved in cardiotoxicity and elucidate the underlying regulatory mechanisms. By shedding light on the interactions between lncRNAs and specific drugs, we hope to pave the way for the development of novel therapeutic targets for the prevention and treatment of drug-induced cardiotoxicity.

Overall, this thesis will contribute to the growing body of knowledge on the role of lncRNAs in disease pathogenesis, particularly in drug-induced cardiotoxicity. Through our research efforts, we aim to provide valuable insights that can inform future studies and potentially lead to the development of innovative therapeutic interventions for this serious medical condition.

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