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Introduction

HIV infection continues to be a major global health concern, with millions of people worldwide living with the virus. Despite significant advancements in antiretroviral therapy (ART), the development of drug resistance remains a major obstacle in the effective treatment of HIV. Autophagy, a cellular process involved in the degradation and recycling of cellular components, has been implicated in various diseases, including infectious diseases such as HIV. However, the role of autophagy in the development of drug resistance in HIV infection remains poorly understood.

This thesis aims to explore the role of autophagy in the development of drug resistance in HIV infection. By investigating the mechanisms by which autophagy influences drug resistance in HIV-infected cells, this study seeks to provide insights into potential therapeutic strategies to overcome drug resistance in HIV treatment.

Chapter 1: Introduction
1.1 Introduction
1.2 Background of study
1.3 Problem Statement
1.4 Objective of study
1.5 Limitation of study
1.6 Scope of study
1.7 Significance of study
1.8 Structure of the Thesis
1.9 Definition of terms

Chapter 2: Literature Review
2.1 Overview of HIV infection and drug resistance
2.2 Autophagy: mechanisms and regulation
2.3 Role of autophagy in infectious diseases
2.4 Autophagy and drug resistance in cancer
2.5 Autophagy and drug resistance in HIV infection
2.6 Mechanisms of drug resistance in HIV infection
2.7 Targeting autophagy as a therapeutic strategy in HIV treatment
2.8 Current challenges in HIV treatment and drug resistance
2.9 Future directions in autophagy research in HIV infection
2.10 Conclusion

Chapter 3: Research Methodology
3.1 Study design
3.2 Sample selection
3.3 Data collection methods
3.4 Data analysis
3.5 Statistical analysis
3.6 Ethical considerations
3.7 Timeline
3.8 Budget
3.9 Anticipated challenges
3.10 Conclusion

Chapter 4: Discussion of Findings
4.1 Overview of study findings
4.2 Role of autophagy in drug resistance in HIV infection
4.3 Mechanisms of autophagy-mediated drug resistance
4.4 Implications for HIV treatment
4.5 Comparison with existing literature
4.6 Limitations of the study
4.7 Future research directions
4.8 Conclusion

Chapter 5: Conclusion and Summary
5.1 Summary of key findings
5.2 Contributions to the field
5.3 Practical implications
5.4 Recommendations for future research
5.5 Conclusion

Thesis Overview

HIV infection remains a significant global health challenge, with millions of people living with the virus and requiring antiretroviral therapy (ART) for disease management. Despite the effectiveness of ART in suppressing viral replication and improving the quality of life for HIV-infected individuals, the development of drug resistance poses a major threat to the long-term success of treatment programs. Autophagy, a cellular process involved in the degradation and recycling of cellular components, has emerged as a potential player in the development of drug resistance in HIV infection.

This thesis aims to explore the role of autophagy in the development of drug resistance in HIV infection. By reviewing the existing literature on autophagy, HIV infection, drug resistance, and related topics, this study seeks to provide a comprehensive understanding of the mechanisms by which autophagy influences drug resistance in HIV-infected cells. The research methodology section outlines the study design, sample selection, data collection methods, and analysis techniques employed in this study.

The discussion of findings section presents the key findings of the study, including the role of autophagy in drug resistance in HIV infection, the underlying mechanisms of autophagy-mediated drug resistance, and the implications for HIV treatment. The conclusion and summary section summarizes the main findings of the study, highlights the contributions to the field, discusses practical implications, and provides recommendations for future research in this area.

Overall, this thesis aims to contribute to the growing body of knowledge on the role of autophagy in drug resistance in HIV infection and to inform the development of novel therapeutic strategies to overcome drug resistance in HIV treatment.

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